Nandrolone decanoate drug

The drug, although classified as an anabolic, however, androgenic side effects can occur. For example, perhaps increasing the oiliness of the skin, acne, hair growth on the body and face. Therefore, to avoid these unpleasant consequences, it should be strictly comply with the recommendations on the dosage of the drug. Women should be aware of the possible consequences virialization while taking the drug. In this case, they can have unpleasant phenomena such as voice. This coarse with low androgenic activity, androgenic side effects are much greater than threshold testosterone or fluoxymesterone methandrostenolone the. Do not forget that the drug can reduce the production of natural male libido and testosterone in the body. Reductase inhibitors should not be taken in the course nandrolone, as this may result in the exacerbation of side effects and impairment of general physical condition of the athlete.

There are possible estrogenic side effects of Nandrolone despite it not being a very estrogenic hormone, at least not directly. Nandrolone does aromatize slightly. Aromatization refers to the conversion of testosterone to estrogen . This takes place when the testosterone hormone interacts with the aromatase enzyme. When the conversion takes place this can cause estrogen levels to go up, which can promote gynecomastia and water retention. High blood pressure can also become an issue if water retention becomes severe. Along with the low level of aromatase activity Nandrolone is also a progestin and has a strong binding affinity for the progesterone receptor. This may stimulate the mammary tissue and enhance the risk of gynecomastia in sensitive individuals.

Combating the estrogenic side effects of Nandrolone can be achieved by the use of anti-estrogen medications, specifically Aromatase Inhibitors (AI’s) such as Anastrozole ( Arimidex ). Selective Estrogen Receptor Modulators (SERM’s) are also sometimes used, such as Tamoxifen ( Nolvadex ). However, AI’s are the proper choice as they will directly reduce serum estrogen levels and SERM’s will not. An AI should be enough to reduce and avoid gynecomastia unless the individual already has existing gynecomastia that could potentially be exasperated.

Important Note: It’s often been said that Nandrolone based gynecomastia is based on increases in prolactin. It is true that 19-nor steroids can increase prolactin, which can also negatively affect libido and erection function. Some men may need to use a dopamine agonist to combat this. However, it is not prolactin that causes 19-nor based gynecomastia but rather the imbalance between estrogen and progesterone. If you merely combat prolactin you may find yourself with the very gynecomastia you tried to avoid.
 

Even if a drug is rated low in estrogenic activity, unwanted and often unpleasant side effects are possible.
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  A number of factors can influence how any individual person reacts to the androgenic and anabolic effects of an anabolic steroid:

Because nandrolone is not broken down into DHT, the deleterious effects common to most anabolic steroids on the scalp, skin, and prostate are lessened to a degree; but is rather broken down to the much weaker androgen dihydronandrolone. The lack of alkylation on the 17α-carbon drastically reduces the drug’s liver toxicity. Estrogenic effects resulting from reaction with aromataseare also reduced due to lessened enzyme interaction, but effects such as gynaecomastia and reduced libido still occur in larger doses because of other mechanisms. Other side-effects of abuse can include erectile dysfunction and cardiovascular damage, as well as several ailments resulting from the drug’s effect of lowering levels of luteinizing hormone through negative feedback. Erectile dysfunction is attributed to the weaker action of dihydronandrolone in the penis since dihydrotestosterone is a known sexual modulator.

Nandrolone decanoate drug

nandrolone decanoate drug

Because nandrolone is not broken down into DHT, the deleterious effects common to most anabolic steroids on the scalp, skin, and prostate are lessened to a degree; but is rather broken down to the much weaker androgen dihydronandrolone. The lack of alkylation on the 17α-carbon drastically reduces the drug’s liver toxicity. Estrogenic effects resulting from reaction with aromataseare also reduced due to lessened enzyme interaction, but effects such as gynaecomastia and reduced libido still occur in larger doses because of other mechanisms. Other side-effects of abuse can include erectile dysfunction and cardiovascular damage, as well as several ailments resulting from the drug’s effect of lowering levels of luteinizing hormone through negative feedback. Erectile dysfunction is attributed to the weaker action of dihydronandrolone in the penis since dihydrotestosterone is a known sexual modulator.

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